Biophysical properties of human b-cardiac myosin with converter mutations that cause hypertrophic cardiomyopathy

نویسندگان

  • Masataka Kawana
  • Saswata S. Sarkar
  • Shirley Sutton
  • Kathleen M. Ruppel
  • James A. Spudich
چکیده

Department of Biochemistry, Stanford University School of Medicine, Stanford, CA 94305, USA. Department of Medicine, Division of Cardiovascular Medicine, Stanford University School of Medicine, Stanford, CA 94305, USA. Department of Pediatrics (Cardiology), Stanford University School of Medicine, Stanford, CA 94305, USA. *Corresponding author. Email: [email protected] (J.A.S.); kruppel@stanford. edu (K.M.R.)

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منابع مشابه

Biophysical properties of human β-cardiac myosin with converter mutations that cause hypertrophic cardiomyopathy

Hypertrophic cardiomyopathy (HCM) affects 1 in 500 individuals and is an important cause of arrhythmias and heart failure. Clinically, HCM is characterized as causing hypercontractility, and therapies are aimed toward controlling the hyperactive physiology. Mutations in the β-cardiac myosin comprise ~40% of genetic mutations associated with HCM, and the converter domain of myosin is a hotspot f...

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تاریخ انتشار 2017